Intravitreal triamcinolone acetonide as treatment of ischemic ophthalmopathy.

PURPOSE: To describe the clinical course of a patient receiving repeated intravitreal injections of triamcinolone acetonide (25 mg) as treatment of ischemic ophthalmopathy. METHODS: A 70-year-old patient with Waldenstrom disease presented with progressive iris neovascularization, vitreous hemorrhage, and ocular hypotony due to ischemic ophthalmopathy. Visual acuity was 0.05. Within 2.5 years, he received three intravitreal injections of 25 mg of triamcinolone acetonide. Additionally, penetratingkeratoplasty and synechiolysis were performed during the follow-up. RESULTS: After each intravitreal injection, visual acuity and intraocular pressure increased, iris neovascularization regressed, and vitreous haze cleared up. CONCLUSIONS: Intravitreal triamcinolone acetonide may induce regression of iris neovascularization, increase intraocular pressure, and improve visual acuity in eyes presenting with ocular hypotony, vitreous hemorrhage, and progressive intraocular neovascularization due toischemic ophthalmopathy.

作 者：

Jonas JB；Kreissig I；Degenring RF

刊 名：

European journal of ophthalmology 

年，卷(期)：

2003V.13(no.6) 

分类号：

 

关键词：

Triamcinolone Acetonide  Pathologic Neovascularization  Visual Acuity  therapeutic aspects  曲安奈德  视敏度

正文语种：

eng 

基金项目：